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Don't create your own pdf viewer . Users just need the Adobe Reader plug in installed on their browser. If you create a custom solution, you ... following his description of visual hallucinations in a sane person The topic of senile hallucinosis has been reviewed by Gold and Rabin, and 60 such patients were reported in detail by Teunisse and colleagues The latter authors found that 11 percent of older persons with reduced vision experienced these phenomena at one time or another In the majority of their cases the hallucinations lacked any personal meaning and were not distressing, but the patients were greatly relieved to hear that they did not have mental disease Curiously, there are reports of improvement in the symptoms with the use of anticonvulsants such as gabapentin (Paulig and Mentrup) Further comments are to be found on page 220 It has been traditionally taught that the lesions responsible for visual hallucinations, if identi able, are situated in the occipital lobe or posterior part of the temporal lobe and that elementary hallucinations have their origin in the occipital cortex and complex ones, in the temporal cortex However, the opposite may pertain; in some cases, formed hallucinations are related to lesions of the occipital lobe and unformed ones to lesions of the temporal lobe (Weinberger and Grant) Also, as emphasized by these authors, lesions that give rise to visual hallucinations, simple or elaborate, need not be con ned to central nervous structures but may be caused by lesions at every level of the neuro-optic apparatus (retina, optic nerve, chiasm, etc) The hallucinatory phenomena of delirium are nonlocalizable, as pointed out in Chap 20, but sometimes the evidence points to an origin in the temporal lobe The Visual Agnosias Visual Object Agnosia This rare condition, rst described by Lissauer in 1890, consists of a failure to name and indicate the use of a seen object by spoken or written word or by gesture The patient cannot even tell the generic class of the object presented Visual acuity is intact, the mind is clear, and the patient is not aphasic conditions requisite for the diagnosis of agnosia If the object is palpated, it is recognized at once, and it can also be identi ed by smell or sound if it has an odor or makes a noise Moving the object or placing it in its customary surroundings facilitates recognition In most reported instances of object agnosia, the patient retains normal visual acuity but cannot identify, match, or name objects presented in any part of the visual elds; if misnamed, the object is used in a fashion that re ects the incorrect perception Amazingly, one encounters patients who have lost the capacity to recognize only one class of objects eg, animals or vegetables suggesting that, in the human, stored information is grouped and classi ed in a way that is necessary for visual perception (we are unable to corroborate this from our own material) Lissauer conceived of visual object recognition as consisting of two distinct processes the construction of a perceptual representation from vision (apperception) and the mapping of this perceptual representation onto stored percepts or engrams of the object s functions and associations and he proposed that impairment of either of these processes could give rise to a defect in visual object recognition As indicated in Chap 13, visual object agnosia is usually associated with visual verbal agnosia (alexia) and homonymous hemianopia Prosopagnosia (the inability to identify faces; see further on) is also present in most cases The underlying lesions are usually bilateral, although McCarthy and Warrington have related a case to a restricted lesion of the left occipitotemporal region (by MRI) Two of our patients with visual object agnosia had an incomplete amnesic syndrome from a left-sided inferior occipital and. birt ean 13, birt report barcode font, birt gs1 128, birt code 128, birt upc-a, birt data matrix mvc display pdf in browser Asp. Net MVC pdf viewer - CodeProject
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