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may make the two dif cult to distinguish As with the related frontal gait, described below, patients who have dif culty initiating gait or whose steps are so short as to be ineffective are helped by marching to a cadence or in step with the examiner In patients with untreated NPH, one observes a progressive deterioration of stance and gait from an inability to walk to an inability to stand, sit, and rise from or turn over in bed There is no hint of ataxia, but a study of the mechanics of the gait in NPH by Stolze and colleagues described a widened base and slight outward rotation of the feet Also distinguishing the gait of NPH from that of Parkinson disease in their study was a response in the latter to acoustic and visual cues for cadence Our own experience is that both types may improve when a cadence is provided

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Standing and walking may be severely disturbed by diseases that affect the frontal lobes, particularly their medial parts and their connections with the basal ganglia This disorder is sometimes spoken of as a frontal lobe ataxia or as an apraxia of gait among numerous other labels, since the dif culty in walking cannot be accounted for by weakness, loss of sensation, cerebellar incoordination, or basal ganglionic abnormality Whether the gait disorder should be designated as an apraxia, in the sense of Liepmann s concept of arm and hand apraxia, is questionable, since walking is instinctual and not learned Patients with so-called apraxia of gait do not have apraxia of individual limbs, particularly of the lower limbs; conversely, patients with apraxia of the limbs usually walk normally More likely, the disorder represents a loss of integration, at the cortical and basal ganglionic levels, of the essential instinctual elements of stance and locomotion that are acquired in infancy and often lost in old age As a shorthand, they are listed here as frontal ; in any case, most cases of central gait disorder are accompanied by a degree of frontal lobe dementia Patients assume a posture of slight exion with the feet placed farther apart than normal They advance slowly, with small, shuf ing, hesitant steps At times they halt, unable to advance without great effort, although they do much better with a little assistance or with exhortation to walk in step with the examiner or to a marching cadence Turning is accomplished by a series of tiny, uncertain steps that are made with one foot, the other foot being planted on the oor as a pivot There is a need to seek support from a companion s arm or nearby furniture The initiation of walking becomes progressively more dif cult; in advanced cases, the patient makes only feeble, abortive stepping movements in place, unable to move his feet and legs forward; in even more advanced cases, the patient can make no stepping movements whatsoever, as though his feet were glued to the oor These late phenomena have been referred to colloquially as magnetic feet or the slipping clutch syndrome (Denny-Brown) and as gait ignition failure (Atchison et al) In some patients, dif culty in the initiation of gait may be an early and apparently isolated phenomenon; but invariably, with the passage of time, sometimes of years, the other features of the frontal lobe gait disorder become evident Finally, as in untreated NPH, these patients become unable to stand or even to sit; without support, they fall helplessly backward or to one side In an attempt to describe these disorders, Liston and colleagues have separated them into three categories: ignition apraxia, disequilibrium, and mixed types They associate trouble starting the gait cycle, shuf ing, and freezing with the rst type and poor balance with the second This is a useful restatement of the phenomenology, but.

This section describes common devices called field-replaceable modules, as well as their role in the computer system as a whole This information will form the foundation of your ability to discover and resolve computer problems If you know the functions of each component, you will more easily be able to determine which component is at fault when something goes wrong

The need for monitoring the hardware and software components comprising the Exadata Database Machine is unquestionable Sometimes, a malfunctioning hardware component or an erratic behavior of a software component can cause failures that can adversely affect performance, or even the availability of the Exadata Database Machine Being able to proactively detect and fix hardware and software issues will ensure optimal performance and healthy functioning of the Database Machine components This section will discuss the different tools and utilities available to monitor the Exadata Storage Servers The monitoring methods will ensure that the different software and hardware components of the Exadata Storage Servers are operating as advertised and, more importantly, performing to their full potential without incurring undue bottlenecks Any anomalies in the behavior of these components will be captured by the monitoring infrastructure, and the administrators will be alerted so that the corrective actions can be immediately implemented The tools available for monitoring the Exadata Storage Servers are the command-line utilities such as CellCLI and SQLPlus, and the web-based Oracle Enterprise Manager The following options for monitoring the Exadata Storage Servers are discussed in this section:

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