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Mercury poisoning arises in two forms, one due to inorganic compounds (elemental or mercury salt) and the other, more dangerous, to organic mercury The sources of potential exposure are reviewed by Clarkson and colleagues Among the organic compounds, methyl mercury gives rise to a wide array of serious neurologic symptoms that may be delayed for days or weeks after exposure, including tremor of the extremities, tongue, and lips; mental confusion; and a progressive cerebellar syndrome, with ataxia of gait and arms, intention tremor, and dysarthria Choreoathetosis and parkinsonian facies have also been described Changes in mood and behavior are prominent, consisting at rst of subjective weakness and fatigability and later of extreme depression and lethargy alternating with irritability This delayed form of subacute mercury poisoning has been reported in chemical laboratory workers after exposure to methyl mercury compounds These agents, particularly dimethyl mercury, are extremely hazardous because they are absorbed transdermally and by inhalation, allowing severe toxicity to occur with even brief contact In a fatal case of a chemist reported by Nierenberg et al, a rapidly progressive ataxia and stupor progressing to coma developed 154 days after exposure Cerebellar function was most severely impaired, and visual function was affected The pathologic changes are characterized by a striking degeneration of the granular layer of the cerebellar cortex, with relative sparing of the Purkinje cells and neuronal loss and gliosis of the calcarine cortex and to a lesser extent of other parts of the cerebral cortex similar to the Minamata cases described later The chronic form of inorganic mercury poisoning occurs in persons exposed to large amounts of the metal used in the manufacture of thermometers, mirrors, incandescent lights, x-ray machines, and vacuum pumps Since mercury volatilizes at room temperature, it readily contaminates the air and then condenses on the skin and respiratory mucous membranes Nitrate of mercury, used formerly in the manufacture of felt hats ( mad hatters ), and phenyl mercury, used in the paper, pulp, and electrochemical industries, are other sources of intoxication Paresthesias, lassitude, confusion, incoordination, and intention tremor are characteristic, and, with continued exposure, a delirious state occurs Headache, various bodily pains, visual and hearing disorders, and corticospinal signs may be added, but their pathologic basis is unknown The term erethism was coined to describe the timidity, memory loss, and insomnia that were said to be characteristic of chronic intoxication If the exposure is more than a minimal degree over a long period, gastrointestinal disturbances are prone to occur (anorexia, weight loss), as well as stomatitis and gingivitis with loosening of the teeth Acute exposure to inorganic mercury in larger amounts is even more corrosive to the gastrointestinal system and produces nausea, vomiting, hematemesis, abdominal pain, and bloody diarrhea as well as renal tubular necrosis Isolated instances of polyneuropathy associated with exposure to mercury have also been reported (Albers et al, Agocs et al) and may be responsible for the paresthesias that accompany most cases as well as the acrodynic syndrome described below The polyneuropathy associated with mercury poisoning is discussed on pages 1130 and 1132 The inhalation of vaporized mercury as a result of extensive dental work, or simply the presence of a large number of llings ( amalgam illness ), is alleged to affect the peripheral nerves or to cause fatigue, but the connection is highly doubtful The presence of mercury in industrial waste has contaminated many sources of water supply and sh, which are ingested by hu-. display pdf in iframe mvc MVC To PDF | Convert Files Easily In C# | Iron PDF
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26 Apr 2017 ... Code for MVC looks like this: ... NET MVC version of the code can be found here: ... PdfSharp.PdfGenerator. GeneratePdf ( html , PdfSharp.PageSize.A4); pdf . mans and cause mercurial poisoning So-called Minamata disease is a case in point Between 1953 and 1956, a large number of villagers living near Minamata Bay in Kyushu Island, Japan, were af icted with a syndrome of chronic mercurialism, traced to the ingestion of sh that had been contaminated with industrial wastes containing methyl mercury Concentric constriction of the visual elds, hearing loss, cerebellar ataxia, postural and action tremors, and sensory impairment of the legs and arms and sometimes of the tongue and lips were the usual clinical manifestations The syndrome evolved over a few weeks Pathologically there was diffuse neuronal loss in both cerebral and cerebellar cortices, most marked in the anterior parts of the calcarine cortex and granule cell layer of the cerebellum CT scans in survivors, years after the mass poisoning, disclosed bilaterally symmetrical areas of decreased attenuation in the visual cortex and diffuse atrophy of the cerebellar hemispheres and vermis, especially the inferior vermis (Tokuomi et al) A painful neuropathy of children (acrodynia) has been traced to mercury exposure from interior latex paint, to calomel (mercurous chloride), to teething powders, and to a mercuric fungicide used in washing diapers (Agocs et al, Clarkson) Albers and colleagues observed the appearance of symptoms (mild decrease in strength, tremor, and incoordination) 20 to 35 years after exposure to elemental mercury These authors believed that the natural neuronal attrition with aging had unmasked the neurologic disorder, a theory which we cannot validate Treatment In the treatment of chronic mercury poisoning, penicillamine has been the drug of choice, since it can be administered orally and appears to chelate mercury selectively, with less effect on copper, which is an essential element in many metabolic processes Dimercaptosuccinic acid (succimer), which is also given orally and has few side effects, will probably prove to be a superior form of treatment (Clarkson) Because it increases the concentration of mercury in the brain, BAL is an unsuitable chelating agent. asp.net mvc generate pdf report [PDF] Pro ASP.NET MVC 5
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To serve a PDF document in ASP.Net MVC requires generating a FileResult method. With IronPDF you can use mvc to return a pdf file. This method may then be served by your controller. For a more advanced example you might use your HTML View to generate a HTML string and then convert it to PDF as above. next These lines are the electronic circuits through which signals travel from one component to another and are collectively called the bus Nervous system function may be deranged as part of acute and frequently fatal poisoning with inorganic phosphorus compounds (found in rat poisons, roach powders, and match heads) More important clinically is poisoning with organophosphorus compounds, the best known of which is triorthocresyl phosphate (TOCP) Organophosphates are widely used as insecticides Since 1945, approximately 15,000 individual compounds in this category have come into use Certain ones, such as tetraethylpyrophosphate, have been the cause of major outbreaks of neurologic disorder, especially in children These substances have an acute anticholinesterase effect but no delayed neurotoxic action Chlorophos, which is a 1-hydroxy-2,2,2-trichlorethylphosphonate, is an exception; it has both an acute and delayed action, as does TOCP The immediate anticholinesterase effect manifests itself by headache, vomiting, sweating, abdominal cramps, salivation, wheezing (secondary to bronchial spasm), miosis, and muscular weakness and twitching Most of these symptoms can be reversed by administration of atropine and pralidoxine The delayed effect manifests 2 to 5 weeks following acute organophosphorus insecticide poisoning This takes the form of a distal symmetrical sensorimotor (predominantly motor) polyneuropathy, progressing to muscle atrophy (see Chap 46) Recovery occurs to a variable degree, and then, in patients poisoned with TOCP, signs of cortico- . export to pdf in mvc 4 razor The C# PDF Library - NuGet Must Haves
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