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optokinetic nystagmus cannot be elicited Visual imagination and visual imagery in dreams are preserved With rare exceptions, no cortical potentials can be evoked in the occipital lobes by light ashes or pattern changes (visual evoked response), and the alpha rhythm is lost in the electroencephalogram (EEG) (see Chap 2) Less complete bilateral lesions leave the patient with varying degrees of visual perception There may also be visual hallucinations of either elementary or complex types The mode of recovery from cortical blindness has been studied carefully by Gloning and colleagues, who describe a regular progression from cortical blindness through visual agnosia and partially impaired perceptual function to recovery Even with recovery, the patient may complain of visual fatigue (asthenopia) and dif culties in xation and fusion The usual cause of cortical blindness is occlusion of the posterior cerebral arteries (most often embolic) or the equivalent, occlusion of the distal basilar artery The infarct may also involve the mediotemporal regions or thalami, which share the posterior cerebral artery supply, with a resulting Korsakoff amnesic defect and a variety of other neurologic de cits referable to the high midbrain and diencephalon (drowsiness, akinetic mutism; see page 305) Hypertensive, eclamptic, and hypoxic-ischemic encephalopathy, Schilder s disease and other leukodystrophies, Creutzfeldt-Jakob disease, progressive multifocal leukoencephalopathy, cerebral infarctions following cardiac arrest, and in ltrating glioma are other causes of cortical blindness A transitory form of cortical blindness may occur with head injury, migraine, hypertensive encephalopathy, or the antiphospholipid antibody syndrome, as a consequence of intravascular dye injection and as a toxic effect of a number of drugs such as interferon-alpha or cyclosporine In the case of the latter drugs, imaging studies have disclosed a reversible posterior leukoencephalopathy, similar in appearance to what occurs in hypertensive encephalopathy Visual Anosognosia (Anton Syndrome) The main characteristic of this disorder is the denial of blindness by a patient who obviously cannot see The patient acts as though he could see, and in attempting to walk, collides with objects, even to the point of injury He may offer excuses for his dif culties: I lost my glasses, The light is dim, etc, or he may only evince indifference to his loss of sight The lesions in cases of negation of blindness extend beyond the striate cortex to involve the visual association areas Rarely, the opposite condition arises: a patient is able to see small objects but claims to be blind This individual walks about avoiding obstacles, picks up crumbs or pills from the table, and catches a small ball thrown from a distance Damasio suggests that this might be a type of visual disorientation but with suf cient residual visual information to guide the hand, and that the lesion will be in the visual association areas superior to the calcarine cortex Visual Illusions (Metamorphopsias) These may present as distortions of form, size, movement, or color In a group of 83 patients with visual perceptual abnormalities, Hecaen found that 71 fell under one of four headings: deformation of the image, change in size, illusion of movement, or a combination of all three Illusions of these types have been reported with lesions con ned to the occipital lobes but are more frequently due to shared occipitoparietal or occipitotemporal lesions; they are therefore also considered in earlier sections of this chapter as well as in Chaps 13 and 16 The right hemisphere appears to be involved more often than the left Illusions of movement occur more frequently with posterior tem-. asp.net c# view pdf How To Open PDF File In New Tab In MVC Using C# - C# Corner
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7 Mar 2018 ... In this post, we will learn about how to open pdf or other files in a new tab using c# . For this example first, we need to return file from MVC ... poral lesions or seizures, polyopia (one object appearing as two or more objects) more frequently with occipital lesions (it also occurs in hysteria), and palinopsia (perseveration of visual images) with both posterior parietal and occipital lesions Visual eld defects are present in many of the cases In all these conditions the anatomic correlates are imprecise It is likely that an element of cortical vestibular disorder underlies the metamorphosis of parieto-occipital lesions The vestibular and proprioceptive systems are represented in the parietal lobes of each side and the lesions there are probably responsible for misperceptions of movement and spatial relations (The illusion of tilting of the environment or upside-down vision is known to occur with parieto-occipital lesions but occurs more often with abnormalities of the vestibular system) Visual Hallucinations These phenomena may be elementary or complex, and both types have sensory as well as cognitive aspects Elementary (or unformed) hallucinations include ashes of light, colors, luminous points, stars, multiple lights (like candles), and geometric forms (circles, squares, and hexagons) They may be stationary or moving (zigzag, oscillations, vibrations, or pulsations) They are much the same as the effects that Foerster and Pen eld obtained by stimulating the calcarine cortex in a conscious patient Complex (formed) hallucinations include objects, persons, or animals and infrequently, more complete scenes They are indicative of lesions in the visual association areas or their connections with the temporal lobes They may be of natural size, lilliputian, or grossly enlarged With hemianopia, they appear in the defective eld or move from the intact eld toward the hemianopic one The patient may realize that the hallucinations are false experiences or may be convinced of their reality Since the patient s response is usually in accord with the nature of the hallucination, he may react with fear to a threatening vision or casually if its content is benign The clinical setting for the occurrence of visual hallucinations varies The simplest black-and-white moving scintillations are part of migraine Others, some colored, occur as a seizure aura Often they are associated with a homonymous hemianopia, as already indicated Frequently they are part of a confusional state or delirium (Chap 20) Similar phenomena may occur as part of hypnologic hallucinations in the narcolepsy-cataplexy syndrome In the peduncular hallucinosis of Lhermitte, the hallucinations are purely visual, appear natural in form and color sometimes in pastels, move about as in an animated cartoon, and are considered by the patient to be unreal, abnormal phenomena (preserved insight) Ischemia in the territories of the posterior cerebral arteries is the usual cause In our material, hallucinosis of this type (erroneously called peduncular insofar as that term refers to the cerebral peduncle) has been associated mainly with high mesencephalic ischemia, which obviously may also implicate the occipital lobes As indicated above, the hallucinations in this disorder are purely visual; if hallucinations are polymodal, the lesion is always in the occipitotemporal parts of the cerebrum A special syndrome of ophthalmopathic hallucinations occurs in the blind person The visual images may be of elementary or complex type, usually of people or animals, and are polychromic (vivid colors) They may occupy all of the visual eld (in the totally blind person) in the eld of one eye Moving the eyes or closing the affected eye has variable effects, sometimes abolishing the hallucinations A similar phenomenon in elderly patients with partially impaired vision has been called the syndrome of Charles Bonnet,. asp.net pdf viewer AtoZSourceCode: How to open pdf file in new tab in MVC using c#
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